28 Februari 2011

The To Do List: Pasta

Remember the To-Do List? I bet you don't! I wrote a post about the To-Do List back when I had fewer than 20 RSS subscribers. I might have even written that post before I set up my RSS feed!

I am not sure why it took me so long to try making pasta. I grew up eating only homemade pasta. Every few weeks my father would bust out the pasta machine, roll out long strips of dough and turn flour into a meal. But I have never made pasta at home. I think my hesitance to make pasta at home is because I thought I had to have a pasta maker. I felt that some heavy, silvery set of gears that can be vice gripped to the counter top was integral to the process of making pasta. And indeed it is, for some people. But I have no room for bulky machinery. I barely even use my stand mixer because I don't have enough counter space to keep it out, it must stay locked away in the pantry. Then I read this interesting blog post. And I suddenly realized that one could make pasta at home without another piece of one use wonder equipment! Life has opened up to me!
I quickly emailed my father for his recipe. He said he started with 2 cups of flour and four eggs and about a tablespoon of olive oil. So I started there, with a trusted family recipe. Only I made some slight alterations--because you wouldn't expect anything less from me! No really, it is not that bad this time. I just made it different by using whole wheat flour, something my dad never does. I used 100% white whole wheat flour. I remember watching Mario Batali making pasta, and he always made the flour into a pile, and carved out a well. I cracked my eggs in, one, two and three--whoops! My third egg spilled over the wall of my well an all over my counter. Eggs look so solid when they are cracked into a bowl. But they run like water when they have nothing to contain them. Rats! I scooped the egg back into the messy flour. The recipe called for 4 eggs. But I figured that 3 eggs would just make a slightly smaller batch. Plus I was using extra large eggs. I hate that everything seems bigger than natural in the US (even though I love my country--have I mentioned that?). I never trust foods in bigger forms. But recently I discovered that my recipes were just coming out better when I used extra large eggs. I don't know why, but it works on everything from muffins to custards.
Once I got the egg scooped back into the flour, I added some oil and went to town. You want pasta to have some tooth. So I kneaded the dough to develop the gluten. But pasta dough is not like pizza dough. It is thicker and tougher. I continued to add flour until the dough was no longer sticky. I took my ball of dough and broke it into 4 smaller pieces. And then I rolled each piece out in turn. It took significant upper body strength to get each piece of dough to roll out thin enough. But that works out well for me because I haven't hit a real gym in like 7 or 8 years. And I am looking for some compelling upper body workouts that center around my interests. No pain no gain right??
After I got each sheet rolled out I rolled it into a flat packet. And I used a serrated bread knife to cut the roll-up into thin strips. I first tried just a serrated steak knife--But don't do it. The force is better distributed in a longer serrated knife. My strips were smaller and straighter and overall better looking after I switched.
Then, because I have no pasta rack, I dried the strips on a cutting board. I just let them hang out on the board for a couple hours. But then I got freaked out about the raw eggs even though that seemed counter intuitive, and my father has let every batch of pasta air dry for years and years. And I did get the recipe from him! Oh well. I put all the semi dry noodles into a plastic bag and parked them into the fridge.
Make no mistake. These are not your restaurant angel hair noodles. These noodles are thick and not altogether straight. They are brown from the wheat germ, and they felt heavy and toothy.

We didn't eat them right away. We had other wheat dinners and I have this thing about not eating pasta too often. I try to space it out over the course of a week. And I don't think we have ever had pasta more than twice in one week. Not because I am bragging about that. I am a little nuts about the wheat and grains. But I have to explain why I didn't have this post up sooner! Last week it was imperative that I make my friend D a lasagna because she had a baby. Yay! Congrats! And if you are going to the trouble to make one lasagna, you really ought to make two. So we had already made pasta for dinner on Monday, plus leftovers. It was Friday before I was read to have pasta again. And even that I felt like was too many nights eating pasta for dinner. But I took one for the team.

I usually have some frozen spaghetti sauce put up for a weeknight meal. So I brought out my last batch and took out my homemade pasta. It was ugly stuff. A week of drying out in the fridge took it's toll on my precious strippy strips. Then it hit me. I had no idea how much pasta I had made. I usually buy 12 oz boxes of pasta and I know my family will eat about 8-9 ounces, so not quite one package, two-thirds if I don't want any leftovers. But here I had this ziploc bag of homemade pasta, how much should I make??

I brought out the old Weight Watchers scale and laid a plate on top. I measured out 9 ounces of pasta. Judging by what was left I think I made about 11 or 12 ounces of pasta. 9 ounces didn't look like enough. But scales don't lie.
My pasta looked so ragged and handmade (not in the good way). I was scared that it would crumble and fall apart in the water. Some strips had already broken into shorter pieces as I was drying and bagging them. I threw it in the pot and held my breath. I usually boil pasta for 10-12 minutes. I did the same with these bad boys. They did not break, they lolled gently in the water. I tried one to test it, gave them another 2 minutes and drained. Only because they were so thick, the noodles were rather al dente. Too bad I didn't realize this until after I was eating them at the dinner table covered in sauce. Oh well, better luck next time.
The kids adore pasta. And the transition to whole wheat pasta has been far easier than expected. A year ago I swore that I just couldn't do whole wheat pasta. I had tried it, I hated the flavor AND the texture, and that I could do healthy whole grains everywhere else but pasta. Then when we started the blog I started buying only whole grain pasta. Strangely, no one minded. DH didn't bitch at me about the grainy texture. And while the kids definitely looked at me liked I was keeping a secret from them, they ate it. And now they don't know any different. I was also pleased how well they took the next leap from store bought to homemade. Given the difference in my wild free form pasta from the eggless, perfectly extruded stuff they are used to, I was shocked that everyone cleaned their plate. The pasta was really really good. very tasty. But it was anything but soft.

For next time: I will use a little more olive oil to give the dough some conditioning. I will also try and roll the sheets a little thinner before cutting the strips. That's easier said than done because rolling out the sheets was, like I mentioned, like doing upper body work at the gym, but one must strive for perfection. I will also cut the strips thinner. Because if I can only make the sheets so thin with a rolling pin, I can reduce necessary cooking time by making say, linguine rather than fettuccine or tagliatelle. I don't even know what I did. Some of my strips were tagliatelle, some were like lasagna noodles (just kidding).
I will also try and make this pasta in larger batches. No joke, it was quick. It took me about 45 minutes from when the first pile of flour hit the counter to when I was cleaned up and the noodles were drying. But I don't really want to have to do that every week. Once a month would be more doable on my schedule. Maybe this will be a good opportunity to commit to eating pasta less? I like that my pasta has many eggs in it. So it is higher in protein than the store bought stuff. But it is still a grain based meal, very filling and lower in protein. While we ate alot of pasta last week, we usually have it only once a week. Perhaps now I could try and get down to once every other week? It is a thought. I am not opposed to grains in a general sense. But while I know that they have their place in my diet (I really do feel better and more energetic when I eat some every day), grains like to take over. They can be addictive. I have to keep them in their place, so to speak.

And finally, let me say that I am proud of this experience. While we are getting really comfortable finding what 'real food' options work for us on a busy working parent schedule, I love challenging one more convenience food. I can now take flour and eggs and turn it into pasta. Take THAT food industry. That's one more thing I don't need you for!

Ready, Set ...

Bloom!  I went to Macon yesterday for a conference and was surprised at all the plants blooming along the roads.  I must live in a cold pocket - elsewhere obviously Spring is upon us!  Branches glowing with a deep red mist belong to our native red maple (Acer rubrum), one of the first native trees to flower.  Other trees sported greenish branches as they began to leaf out ahead of their flowers.

Acer rubrum flowers just emerging


In Georgia we are fortunate to be able to celebrate spring a little earlier than some parts of the country – Hepatica has been blooming at my house for several weeks, and I found my first trout lily bloom this week (and by the end of the week there were many more than that first one). 

But don’t let these early herbaceous blooms distract you from the lengthening of woody buds and the emergence of those first few tender leaves on our trees and shrubs. I find the process of growth so much more interesting on these plants – perhaps their branches offer a most visible window into the process of new growth.  Tightly packed buds swell and lengthen, causing the bud scales to fall away and allowing the new growth to emerge.  Tiny leaves, perfect miniatures of their mature forms, begin the process of capturing sunlight and making food for the plant.  In some cases, of course, flowers may emerge first, feeding those first insects whose life-cycle has evolved to be there just at this moment.

On Monday of this week I discovered these early leaves on the crabapples (Malus angustifolia).  Just a few days before, these were sparkling, ruby-colored dots against the brown-grey branches.  Now they are unfolding, first as translucent red leaves and then gradually turning green as they mature and grow.

Malus angustifolia



Cercis canadensis buds
Cercis candadensis blooms













Here is a redbud (Cercis canadensis) flower bud just beginning to open.  In about a month, this tree will be covered with the purplish pea-like flowers that delight us.

Aesculus pavia

Aesculus pavia in 2008













A red buckeye (Aesculus pavia) bud is also swelling with the promise of flowers.  The earliest hummingbirds will appreciate the bright red flowers.




It is time to keep the camera nearby, ready to capture those fleeting blossoms.  The flowers will be coming quickly now.  Photographing them is a great way to remember those beautiful blooms.  It also helps me notice details and keep track of bloom times from year to year.  And, as you can see above, it helps me show you what blooms WILL look like later (those flowering pictures of Redbud and Buckeye are from previous years).

I am still learning about some of the features on my digital camera.  I learned a couple of years ago that I would get better pictures if I’d let the camera do the focusing.  I also realized that using the tiny view finder prevented me from seeing whether the camera had properly focused.  Now I use the big screen on the back of the camera to watch the camera focus; once it is focused on what I want (sometimes it focuses on the wrong area!) then I press the button. 

Digital cameras are great for “do-overs” until you get the picture you want.  Be sure to learn how to review your pictures on site so that you can see if you got the picture you wanted.  You don’t want to figure out that you missed the shot when it is too late to try again.

So get out there and take some pictures.  I will be doing the same and hope to share some of my “good ones” with you in the months to come.

P.S. That bright white tree blooming on the side of the road is not necessarily something to admire.  I saw a lot of escaped ‘Bradford’ pears on I-75 yesterday.  These are thorny trees that grew from the fruit that some ornamental pears are developing lately.  They bloom earlier than any of our native white flowering trees and usually have a very upright shape.  You can read more about this escaped plant in the delightfully informative article “Who Let the Pears Out?”.

There is No February 29th This Year

Sorry for the two tasty previews. There is, apparently, no 2/29/11 this year. And when you type in an invalid date, Blogger simply publishes your post. Nice, right?

So sorry for letting the cat out of the bag. Hope you will come back and enjoy that post on 3/1. Since there is a 3/1 this year (and every year).

Black Cumin and Pancreatic Cancer

UPDATE: Feb 2011 - Gernod Katzer's Spice pages are a resource I frequently use  to help people learn more about the benefits of herbs for health.  Here is the page on Nigella sativa

Original post date April 2009

For Black oil purchases please contact us.

See also enzymes for pancreatic cancer and other related articles here on Natural Health News (search window)
DENVER, April 21 (UPI) -- An anti-inflammatory herbal extract may inhibit pancreatic cancer, U.S. researchers say.

Thymoquinone -- the major constituent of the oil extract from a Middle Eastern herbal seed called Nigella sativa used as a traditional medicinal in Middle Eastern and Asian countries -- reduces the release of inflammatory mediators in pancreatic cancer cells.

Dr. Hwyda Arafat of the Jefferson Medical College of Thomas Jefferson University in Philadelphia, who was the study leader, says thymoquinone almost completely abolished the expression of several inflammatory cytokines, including: TNF-alpha, interleukin-1beta, interleukin-8, Cox-2 and MCP-1.

This effect was superior to the effect of trichostatin A -- an inflammation inhibitor already shown to ameliorate inflammation-associated cancers.

The herb also inhibited the activation and synthesis of NF-kappaB, a transcription factor that has been implicated in inflammation-associated cancer. Activation of NF-kappaB has been observed in pancreatic cancer and may be a factor in pancreatic cancer's resistance to chemotherapeutic agents.

"These are very exciting and novel results," Arafat said in a statement. "These potent effects show promise for the herb as a potential preventive and therapeutic strategy for pancreatic cancer. More importantly, the herb and oil are safe when used moderately, and have been used for thousands of years without reported toxic effects."

The findings are being presented at the American Association for Cancer Research's 100th annual meeting in Denver.
Clinical Data on Black Seed Herb

Black seed is used for treating gastrointestinal conditions including gas, colic, diarrhea, dysentery, constipation and haemorrhoids. It is also used for respiratory conditions, including asthma, allergies, cough, bronchitis, emphysema, flu and congestion. Additionally, it is used as an antihypertensive, immunoprotectant, anticancer agent, and vermifuge. It is used orally for women's health: including as a contraceptive, for stimulation of menstruation, and increasing milk flow.

In allergic conditions, black seed may have antihistamine effects. Black seed is thought to have immunoprotectant effects. Preliminary evidence suggests it may help minimize chemotherapy-induced decreases in hemoglobin and leukocyte counts. Black seed may be beneficial as an anticancer agent. According to preliminary studies, black seed may inhibit stomach tumors, carcinoma, and Ehrlich ascites carcinoma.

Black seed is reported to have been used for over 2000 years. Recordings mention it as far back as 1400 years. Black seed was found in the tomb of King Tutankhamen.

Published Clinical Studies
Black Seed Herb

The in vitro effect of aqueous extract of Nigella sativa seeds on nitric oxide production.(click here to view report)

Neuropharmacological activity of Nigella sativa L. extracts.(Click here to view)

Chemopreventive potential of volatile oil from black cumin (Nigella sativa L.) seeds against rat colon carcinogenesis.(Click here to view)

Nigella sativa (black cumin) ameliorates potassium bromate-induced early events of carcinogenesis: diminution of oxidative stress.(Click here to view)

Delayed apoptosis upon the treatment of Hep-2 cells with black seed.(Click here to view)

Effect of Nigella sativa (N. sativa L.) and oxidative stress on the survival pattern of MCF-7 breast cancer cells.(Click here to view)

Protective effect of Nigella sativa seeds against carbon tetrachloride-induced liver damage.(Click here to view)

Effect of Nigella sativa (black seed) on subjective feeling in patients with allergic diseases.(Click here to view)

The in vitro effect of aqueous extract of Nigella sativa seeds on nitric oxide production.

Mahmood MS, Gilani AH, Khwaja A, Rashid A, Ashfaq MK.

Department of Biological and Biomedical Sciences, The Aga Khan University Medical College, Karachi, Pakistan.

The in vitro effect of aqueous extract of Nigella sativa seeds on nitric oxide (NO) production by murine macrophages was studied. Murine peritoneal macrophages were pre-incubated with the extract and then activated with Escherichia coli lipopolysaccharride. NO production was measured after 24 hours by spectrophotometry. The plant extract caused a dose-dependent decrease in NO production. Dialyzed preparation of the extract did not affect NO production. However, the boiled fraction of the extract resulted in a dose-dependent inhibition of NO apparently comparable to that of the whole extract. These results indicate that the aqueous extract of N. sativa seeds exhibits an inhibitory effect on nitric oxide production by murine macrophages and the active component(s) is/are non-protein in nature. In view of the fact that nitric oxide is a pro-inflammatory mediator, this study validates the traditional use of the Nigella sativa seeds for the treatment of rheumatism. Copyright 2003 John Wiley & Sons, Ltd.

PMID: 13680825 [PubMed - indexed for MEDLINE]

Neuropharmacological activity of Nigella sativa L. extracts.

Al-Naggar TB, Gomez-Serranillos MP, Carretero ME, Villar AM.

Facultad de Farmacia, Universidad Complutense de Madrid, 28040 Madrid, Spain.

Pharmacological studies have been conducted on the aqueous and methanol extracts of defatted Nigella sativa L. seeds to evaluate their effects on the central nervous system (CNS) and on analgesic activity. The observations suggest that the two extracts of Nigella sativa possesses a potent CNS and analgesic activity (depressant action especially in the case of the methanolic extract).

PMID: 12902052 [PubMed - indexed for MEDLINE]

Chemopreventive potential of volatile oil from black cumin (Nigella sativa L.) seeds against rat colon carcinogenesis.

Salim EI, Fukushima S.

Laboratory of Experimental and Molecular Carcinogenesis, Department of Zoology, Faculty of Science, Tanta University, Tanta 31527-Egypt.

Chemopreventive effects of orally administered Nigella sativa oil on the induction and development of 1,2-dimethylhydrazine-induced aberrant crypt foci (ACF), putative preneoplastic lesions for colon cancer, were investigated in Fischer 344 rats. Starting at 6 wk of age, 45 male rats (groups 1-3) were subcutaneously injected with DMH once a week for 3 wk. Group 1 (15 rats) served as a carcinogen control group without N. sativa administration. Group 2 or 3 (15 rats each) were given the oil in the postinitiation stage or in the initiation stage, respectively. Animals of group 4 (11 rats) were injected with 0.9% saline and received N. sativa oil from the beginning until the termination. At sacrifice, 14 wk after the start, the total numbers of ACF as well as those with at least four crypts were significantly reduced in group 2 (P < 0.01). However, treatment with N. sativa oil in the initiation stage (group 3) did not exhibit significant inhibitory effects except on foci with only one aberrant crypt. Immunohistochemical analysis of 5-bromo-2'.-deoxyuridine labeling in colonic crypts revealed the N. sativa oil to have significant antiproliferative activity in both initiation and postinitiation stages and especially in the latter. Histological examination revealed no pathological changes in the liver, kidneys, spleen, or other organs of rats treated with N. sativa. In addition, biochemical parameters of blood and urine as well as body weight gain were not affected. These findings demonstrate that the volatile oil of N. sativa has the ability to inhibit colon carcinogenesis of rats in the postinitiation stage, with no evident adverse side effects, and that the inhibition may be associated, in part, with suppression of cell proliferation in the colonic mucosa. PMID: 12881014 [PubMed - in process] Nigella sativa (black cumin) ameliorates potassium bromate-induced early events of carcinogenesis: diminution of oxidative stress. Khan N, Sharma S, Sultana S. Section of Chemoprevention and Nutrition Toxicology, Department of Medical Elementology and Toxicology, Faculty of Science, Jamia Hamdard, Hamdard University, New Delhi 110 062, India. Potassium bromate (KBrO3) is a potent nephrotoxic agent. In this paper, we report the chemopreventive effect of Nigella sativa (black cumin) on KBrO3-mediated renal oxidative stress, toxicity and tumor promotion response in rats. KBrO3 (125 mg/kg body weight, intraperitoneally) enhances lipid peroxidation, gamma-glutamyl transpeptidase, hydrogen peroxide and xanthine oxidase with reduction in the activities of renal antioxidant enzymes and renal glutathione content. A marked increase in blood urea nitrogen and serum creatinine has also been observed. KBrO3 treatment also enhances ornithine decarboxylase (ODC) activity and [3H] thymidine incorporation into renal DNA. Prophylaxis of rats orally with Nigella sativa extract (50 mg/kg body weight and 100 mg/kg body weight) resulted in a significant decrease in renal microsomal lipid peroxidation (P < 0.001), gamma-glutamyl transpeptidase (P < 0.001), H2O2 (P < 0.001) and xanthine oxidase (P < 0.05). There was significant recovery of renal glutathione content (P < 0.01) and antioxidant enzymes (P < 0.001). There was also reversal in the enhancement of blood urea nitrogen, serum creatinine, renal ODC activity and DNA synthesis (P < 0.001). Data suggest that Nigella sativa is a potent chemopreventive agent and may suppress KBrO3-mediated renal oxidative stress, toxicity and tumour promotion response in rats. PMID: 12755470 [PubMed - indexed for MEDLINE]
More Info - Clinical Studies - References - Buy Black Seed Oil now
Delayed apoptosis upon the treatment of Hep-2 cells with black seed. Corder C, Benghuzzi H, Tucci M, Cason Z. University of Mississippi Medical Center, Jackson, MS 39216, USA. Nigella sativa (Black seed, BS) has been used to promote health and fight disease for centuries. The objectives of this investigation were: (1) to study whether agents such as cortisol and LPS alone or in combination induce cellular (Hep-2, laryngeal carcinoma) damage with time in culture (24, 48, and 72 hours) using apoptosis as a marker, (2) to determine if an immune stimulant such as BS, can protect Hep-2 cells from insult and ultimately thwart the programmed cells death mechanism. A total of 54 Hep-2 cell/tubes (50,000 cells per tube) were divided into six equal groups. Group one served as untreated control, while groups 2-6 were treated with either cortisol (10 ng/ml), LPS (10 micrograms/ml), BS (25 micrograms/ml), or a combination of LPS and cortisol and cortisol plus LPS plus BS, respectively. At the end of each phase the cells were harvested, heat fixed and stained with H&E to evaluate morphological changes. Immunohistochemistry, using antibodies against caspace-3 to evaluate cells undergoing apoptosis was conducted in all groups. The results of this study showed evidence of cells undergoing apoptosis at different magnitudes in all groups. However, the most dramatic change was seen in groups containing cortisol and LPS alone or in combination. This was supported by the fact that there were several adaptive responses observed in all phases. In addition, the exposure of BS to cells pretreated with cortisol and LPS showed evidence of protection against the progressive apoptosis. PMID: 12724921 [PubMed - indexed for MEDLINE] Effect of Nigella sativa (N. sativa L.) and oxidative stress on the survival pattern of MCF-7 breast cancer cells. Farah IO, Begum RA. Jackson State University, Jackson, MS 39217, USA. N. Sativa L., an oriental spice, has long been used as a natural medicine for treatment of many acute as well as chronic conditions. It has been used in the treatment of diabetes, hypertension, and dermatological conditions. There has been very few studies on the effects of N. Sativa as cancer prevention/therapy. Our objective therefore, was to expose MCF-7 breast cancer cells to aqueous and alcohol extracts and in combination with H2O2 as an oxidative stressor. Measurement of cell survival under various concentrations and combinations was conducted using standard cell culture techniques, exposure protocols in 96 well plates and Fluoro-spectrosphotometry. Following cellular growth to 90% confluency, exposure to water (WE) and ethanol (AE) extracts of N. sativa and H2O2 was performed. Toxicity index (LC50) was calculated from percent survival using regression analysis. Results showed that the alcohol extract and its combinations were able to completely inactivate the MCF-7 cells (LC50 ranged from 377.16-573.79 in descending potency for H2O2 + AE, AE and Mix of WE and AE). H2O2 alone effectively inactivated MCF-7 cells (LC50 = 460.94). The least effective combinations in descending potency were WE + H2O2, WE + AE + H2O2, and WE (LC50 were 725.79, 765.94, and 940.5 respectively. Combinations other than AE + H2O2 showed possible interactions, which lead to reduction in their potency. In conclusion, N. Sativa alone or in combination with oxidative stress were found to be effective in vitro in inactivating MCF-7 breast cancer cells, unveiling opportunities for promising results in the field of prevention and treatment of cancer. PMID: 12724920 [PubMed - indexed for MEDLINE] Protective effect of Nigella sativa seeds against carbon tetrachloride-induced liver damage. Al-Ghamdi MS. Department of Pharmacology, College of Medicine, King Faisal University, Dammam, Saudi Arabia. mghamdi@Dammam.kfu.edu.sa It has been reported that Nigella sativa oil possesses hepatoprotective effects in some models of liver toxicity. However, it is N. sativa seeds that are used in the treatment of liver ailments in folk medicine rather than its oil. Therefore, the aim of this study was to investigate the effect of the aqueous suspension of N. sativa on carbon tetrachloride (CCL4)-induced liver damage. Aqueous suspension of the seeds was given orally at two dose levels (250 mg/kg and 500 mg/kg) for five days. CCL4 (250 microl/kg intraperitoneally / day in olive oil) was given to the experimental group on days 4 and 5, while the control group was only treated with the vehicles. Animals treated with CCL4 showed remarkable centrilobular fatty changes and moderate inflammatory infiltrate in the form of neutrophil and mononuclear cells when compared to the controls. This effect was significantly decreased in animals pretreated with N. sativa. Histopathological or biochemical changes were not evident following administration of N. sativa alone. Serum levels of aspartic transaminase (AST), and L-alanine aminotransferase (ALT) were slightly decreased while lactate dehydrogenase (LDH) was significantly increased in animals treated with CCL4 when compared to the control group. LDH was restored to normal but ALT and AST levels were increased in animals pretreated with N. sativa. In conclusion, N. sativa seeds appeared to be safe and possibly protective against CCL4-induced hepatotoxicity. However, further studies may still be needed prior to supporting its use in folk medicine for hepatic diseases. PMID: 14696675 [PubMed - in process] Effect of Nigella sativa (black seed) on subjective feeling in patients with allergic diseases. Kalus U, Pruss A, Bystron J, Jurecka M, Smekalova A, Lichius JJ, Kiesewetter H. Humboldt University, Berlin, School of Medicine, Institute for Transfusion Medicine, Charite University Hospital, Berlin, Germany. Nigella sativa (black seed) is an important medicinal herb. In many Arabian, Asian and African countries, black seed oil is used as a natural remedy for a wide range of diseases, including various allergies. The plant's mechanism of action is still largely unknown. Due to the lack of study data on its efficacy in allergies, four studies on the clinical efficacy of Nigella sativa in allergic diseases are presented. In these studies, a total of 152 patients with allergic diseases (allergic rhinitis, bronchial asthma, atopic eczema) were treated with Nigella sativa oil, given in capsules at a dose of 40 to 80 mg/kg/day. The patients scored the subjective severity of target symptoms using a predefined scale. The following laboratory parameters were investigated: IgE, eosinophil count, endogenous cortisol in plasma and urine, ACTH, triglycerides, total cholesterol, LDL and HDL cholesterol and lymphocyte subpopulations. The score of subjective feeling decreased over the course of treatment with black seed oil in all four studies. A slight decrease in plasma triglycerides and a discrete increase in HDL cholesterol occurred while the lymphocyte subpopulations, endogenous cortisol levels and ACTH release remained unchanged. Black seed oil therefore proved to be an effective adjuvant for the treatment of allergic diseases. Copyright 2003 John Wiley & Sons, Ltd. PMID: 14669258 [PubMed - in process]

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References Hanafy MS, Hatem ME. Studies on the antimicrobial activity of Black seed seed (black cumin). J Ethnopharmacol 1991;34(2-3):275-8. The Review of Natural Products by Facts and Comparisons. St. Louis, MO: Wolters Kluwer Co., 1999. Akhtar MS, et al. Field trial of Saussurea lappa roots against nematodes and Nigella sativa seeds against cestodes in children. JPMA J Pak Med Assoc 1991 Aug;41(8)185-7. Tennekoon KH, Jeevathayaparan S, Kurukulasooriya AP, Karunanayake EH. Possible hepatotoxicity of Nigella sativa seeds and Dregea volubilis leaves. J Ethnopharmacol 1991;31(3):283-9. Keshri G, Singh MM, Lakshmi V, Kamboj VP. Post-coital contraceptive efficacy of the seeds of Black seed in rats. Indian J Physiol Pharmacol 1995;39(1):59-62. Aqel M, Shaheen R. Effects of the volatile oil of Black seed seeds on the uterine smooth muscle of rat and guinea pig. J Ethnopharmacol 1996;52(1):23-6. Nagi MN, Alam K, Badary OA, et al. Thymoquinone protects against carbon tetrachloride hepatotoxicity in mice via an antioxidant mechanism. Biochem Mol Biol Int 1999;47(1):153-9. Chakravarty N. Inhibition of histamine release from mast cells by nigellone. Ann Allergy 1993;70(3):237-42. Worthen DR, Ghosheh OA, Crooks PA. The in vitro anti-tumor activity of some crude and purified components of blackseed, Black seed L. Anticancer Res 1998;18(3A):1527-32. Daba MH, Abdel-Rahman MS. Hepatoprotective activity of thymoquinone in isolated rat hepatocytes. Toxicol Lett 1998;95(1):23-9. Salomi NJ, Nair SC, Jayawardhanan KK, et al. Antitumour principles from Black seed seeds. Cancer Lett 1992;63(1):41-6. Houghton PJ, Zarka R, de las Heras B, Hoult JR. Fixed oil of Black seed and derived thymoquinone inhibit eicosanoid generation in leukocytes and membrane lipid peroxidation. Planta Med 1995;61(1):33-6. Haq A, Abdullatif M, Lobo PI, et al. Black seed: effect on human lymphocytes and polymorphonuclear leukocyte phagocytic activity. Immunopharmacology 1995;30(2):147-55. Badary OA, Al-Shabanah OA, Nagi MN, et al. Inhibition of benzo(a)pyrene-induced forestomach carcinogenesis in mice by thymoquinone. Eur J Cancer Prev 1999;8(5):435-40. Medenica RD. Use of Black seed to increase immune function. U.S. Patent 5,482,711, issued January 9, 1996. Obtained from US Patent and Trademark Ofc on April 12, 2000. www.uspto.gov/patft/index.htm. Increases immune function. ~ U.S. Patents Sections, Antiviral Agents Bulletin #5,482,711 Black seed stimulates bone marrow and immune cells and raises the interferon production, protects normal cells against cell destroying effects of viruses, destroys tumor cells and raises the number of anti-bodies producing B cells. ~ Cancer Immuno-biology Laboratory ,South Carolina Black seed contains valuable unsaturated fatty acids, for example Linoleic and Gammalinolen acids get into the organism. By that it possible to reach a synthesis of important immune regulating substances derived as from Prostaglandin E1. Linoleic acid stabilizes the cell membranes and Prostaglandin has the effect of inhibiting inflammation. By that the immune reactions are stopped which cause the illnesses and which otherwise could be the start of many chronic illnesses like acne and hayfever right up to cancer. ~ Dr. Peter Schleicher Immunologist, Munich, Germany Black seed proves to have an ant histamine, ant-oxidant, anti-biotic, anti-mycotic and broncho-dilating effect. ~ Study of Black seed oil on humans, American Scientists Black seed is truly a remarkable herb that has been used for over 3000 years. It contains over 100 valuable components. It is a significant source of essential fatty acids, proteins, carbohydrates and other vitamins and minerals. " The seeds are also rich in sterols, especially beta-sitosterol, which is known to have anticarcinogenic activity". ~ Dr. Michael Tierra L.AC. O.M.D Black seed tests prove to be genuine universal remedy. ~ Pharmaceutical newspaper, Wissenschaftlicher Text Black seed is a valuable source of protein, carbohydrates, essential fatty acids, vitamins A, B1, B2, C and niacin as well as minerals such as calcium, potassium, iron, magnesium, selenium, magnesium and zinc. ~ Phytochemicals of Nigella sativa seeds. Food Chemistry

Vaccines for Population Control

A vaccine against HCG (Human Chorionic Gonadotrophin) for stopping pregnancy is under development. 

I'm assuming this would be a permanent sterilization? 

How would you "unvaccinate"? 

HCG is a hormone for fat regulation too (Homeopathic HCG is used in a currently popular weight loss program). 

"Human chorionic gonadotropin (hCG) is synthesized soon after fertilization and is essential for embryonic implantation. A an anti-hCG vaccine would be ideal for immuno-contraception."

Learn More -  
http://www.sciencedirect.com/science?_ob=ArticleURL&_udi=B6TD4-521MCG3-1&_user=10&_coverDate=03%2F09%2F2011&_rdoc=20&_fmt=high&_orig=browse&_origin=browse&_zone=rslt_list_item&_srch=doc-info%28%23toc%235188%232011%23999709987%232921799%23FLA%23display%23Volume%29&_cdi=5188&_sort=d&_docanchor&_ct=20&_acct=C000050221&_version=1&_urlVersion=0&_userid=10&md5=574fa3f90e112db16b4a18b05e51943a&searchtype=a

Source

26 Februari 2011

Allergy Season: What Makes It Worse


May 2011 - Are the drugs you take for allergy making you gain weight?

Whether you are a Climate Change believer or not, allergies do seem to be coming on earlier in the year, and are causing more problems for more people.

I always advise to look at your environment and your diet as both these areas have a major impact on allergy.  I think it will be interesting to see how the increase in GMO crops and that pollen will start to affect people.

Remember too that cell phone and other wireless devices that are so popular contribute to this problem by creating electrolytically charged pollen and pollutant particles which makes it easier for these to stick to the mucous membrane lining inside your respiratory system.  This finding from a decade ago also shows correlation to increasing asthma rates.

There are several excellent approaches to natural allergy therapy, find out more here -

and if you'd like a personal program designed specifically for you contact us for this clinical service.

A new USDA-led study finds a warming planet makes for more pollen and a longer, more intense allergy season in many parts of the United States.

If you're planning a vacation during prime hay fever season—summer and fall—opt for a spot near water, where pollen counts tend to be lower.
RODALE NEWS, EMMAUS, PA—Given the millions of allergy sufferers held hostage by the drippy noses, burning, watery eyes, and continuous sneezing sessions it induces, ragweed may be one of the most hated plants on the planet. And a new the U.S. Department of Agriculture (USDA)-led study published in the Proceedings of the National Academy of Sciences confirms what many allergy sufferers and allergists have already been noticing—hay fever season caused by ragweed seems to be getting more intense and lasting longer.
The study is the latest to make the connection between climate change and a more potent allergy season. (Allergy-related issues cost the United States about $21 billion a year, so a warming planet affects economics, too.) "The main takeaway is that we are already seeing a significant increase in the season length of ragweed; and that this increase in season length is associated with a greater warming at northern latitudes, consistent with the Intergovernmental Panel on Climate Change projections regarding climate change," explains lead study author Lewis Ziska, PhD, research plant physiologist with USDA's Crop Systems and Global Change Lab.
THE DETAILS: Researchers used ragweed pollen and temperature data recorded between the late 1990s and 2005 in 10 different locations in the U.S. and Canada and found that in all but two of the areas analyzed, the ragweed pollen season increased—in some cases by nearly a month. The lengthening of the allergy season coincides with an increase in warmer, frost-free days. Researchers noticed a general trend—the ragweed allergy season grew longest in the higher latitudes of the northern United States and Canada. Winnipeg, Ontario, allergy sufferers endured a 27-day-longer ragweed pollen season in 2005 compared to just 16 years earlier. In the U.S., Fargo, ND, and Minneapolis, MN, experienced a more than two-week increase in ragweed allergy season, with LaCrosse and Madison, WI, not far behind.
WHAT IT MEANS: Climate change threatens human health in a number of ways, but allergies may be the most immediate, easy-to-recognize ailment thus far. And our increasingly chaotic climate's allergy-accelerating properties are already afflicting millions of people. Ragweed is one of the most common weed allergens, affecting about 10 percent of the population. Among allergy sufferers, nearly a third endure hay fever misery brought on by ragweed pollen. Under normal circumstances, a single ragweed plant creates 1 million pollen grains; but a climate change–charged, more CO2-rich environment boosts that number to upwards of 3 to 4 million pollen grains per plant, according to Clifford Bassett, MD, medical director of Allergy and Asthma Care of New York and a member of the public-education committee at the American College of Allergy, Asthma and Immunology. (Don't your eyes water just thinking of it?)
And scientists are also suspect of other potentially climate change–infused weed species. Ziska says there are concerns that other specific plant allergens are worsening due to climate change. His research group is working with Rutgers University in New Jersey and the Environmental Protection Agency to begin assessing pollen production and season length for other annual weeds like lambsquarters, mugwort, and plaintain, in addition to ragweed.
Consuming less, using less energy, eating organic, and demanding that clean energy subsidies replace incentives to fund dirty fossil fuels like coal, oil, and natural gas that make us sick are all important tactics to help stabilize global climate and protect our health. It's also important to realize that we've already set ourselves up for a lifetime of climate-related problems. Here's how to deal with the allergy aspect as we all work to keep things from getting worse.
Here are some solutions to think about now, before ragweed allergies strike later this year:
• Make sure you're actually allergic to ragweed. It may sound silly, but allergists recommend being tested to confirm you're allergic to what you actually think is making you sneeze. If ragweed is really making your life miserable (the longer you're exposed to the allergen, the worse the symptoms become), consider getting allergy shots. The ongoing climate shift could be a cue to reassess your antiallergy options. "It might make people who previously had mild ragweed seasons to consider interventions they hadn't though of before, like getting ragweed allergy shots," says study coauthor Jay Portnoy, MD, chief of allergy, asthma, and immunology at Children's Mercy Hospitals and Clinics in Kansas City, MO.
• Plan vacations accordingly. For many people, February still marks the cold season, months away from hay fever hell. But take your ragweed allergy into consideration as you plan this year's summer or fall getaway. Dr. Bassett notes that pollen counts are generally lower around water. So if you vacation during prime ragweed season—summer and fall, or year-round in places like Florida or Hawaii—plan some time on the beach or around rivers and lakes for some ragweed relief.
• Create better indoor air. Now's the perfect time to grow your own houseplants for free. They should be flourishing by ragweed season. While houseplants can't rid your air of pollens you're allergic to, certain houseplants can counteract indoor air pollution that further aggravates your allergy problem.

Diabetes Diary

ProvHerbials in Seattle is supporting The Walk with sales of their natural products.  Learn More Here

In support of The Longest Walk III to Reverse Diabetes, especially for our Indian communities and people, Creating Health Institute has developed The Diabetes Diary.

Daily posts are found on our facebook page.

Learn more about The Longest Walk III here - here - here - and here.

As The Walkers come through your area, please welcome them and support their effort.


In the NEWS


Northern Route

Southern Route

24 Februari 2011

Pink Cause Marketing

20 years of Cause Marketing
I started to wonder why corporations, which on every other front care about making money, suddenly “get philanthropy” when it comes to breast cancer. Now I question the motivation of companies who slap the pink ribbon on their products. I feel exploited as a survivor and feel that most companies use the pink ribbon to increase their sales. Source


Selections from Natural Health News


Oct 05, 2009
4 September 2010 - The Pink Cause Marketing is getting underway once again. I just received a spam from a PR firm about a massage clinic in Seattle that would give $15 from each massage to the Komen Foundation. ...

Oct 02, 2010
This is why for more than a decade we have been saying THINK Before You Pink! Women's Health Month is March, and that is not too far away. FDA Warns of Potential of Serious Side Effects with Topical Numbing Agents . ...

October Pink Promotions
Oct 01, 2008
In your effort to not be swayed by massive marketing of pink lids on toxic products join the campaign to stop Yoplait from using rGBH hormone laced milk to make their synthetic goop. Yoplait touts its yogurt as being healthy for women, ...

Splenda is Getting into Pediatrics!

Last week a friend at work told me about a friend who was just in her pediatrician's office. This friend has been reading The Table of Promise practically since inception! (Thank you!) When she went into her Ped's office, she noticed a flyer regarding childhood obesity, a growing problem of grave concern. The flyer said "More and more children are gaining an unhealthy amount of weight. Children who gain too much weight are more likely to have health problems when they get older. They are also more likely to be overweight as adults. What's the goal for overweight children? The goal is for overweight children to slow down the rate of weight gain. This gives their bodies a chance to catch up with their weight as the grow. Doctors want children to get to and maintain a healthy weight."

Apparently, the flyer says that parents can make two simple changes, cut 100 calories from your child's daily diet and add 2,000 steps. I am all for adding steps. But food is the biggest factor in wieght gain, no matter how much you excercise. And the flyer suggests that part of the way one can reduce calories is to replace sugar with splenda. Me, I say preposterous. The flyer also directs patients to go to the splenda website, which I of course did. They have a whole section on simple changes. I am also all for reducing calories. But isn't it easier to switch to plain yogurt rather than one sweetened with splenda? Or ELIMINATE a snack rather than choosing a lower calorie one?

I am not in favor of fake sweeteners and sugar substitutes. There is significant evidence that they trick the body. The sweet taste prepares the digestive system into believing that it will be soon working on digesting lots of calories. Then like an out of work union, when no calories arrive in your tummy your digestive system lobbies for more work. Your body demands that you eat more since you gyped it out of once promised calories. Not to mention the problems with aspartame, and the problems with a new sweetner neotame. I don't see the same smoking guns for splenda (sucralose) or xylitol based off what I have googled on the internet, but they are chemicals none the less, and I don't to eat them. And the tricks they play on our body are the same.

But the idea that splenda is now infiltrating pediatricians' office in the hopes of plugging their product to overweight kids and desperate parents across the country. I say WRONG WRONG WRONG! Doctors should be prescribing real food to their patients, period. And we parents need to stand up to our kids. We are in charge. If you don't want your kid to eat crap, don't give it to them.

This post is entered in Food Renegade's Fight Back Fridays and Real Food Whole Health's Blog Carnival!

Reducing Fat Leads to Obesity

A new study examining the dietary habits of Americans over a 25-year period has revealed that while overall fat intake reduced by 3%, obesity in men tripled and in women doubled. The research – published this month in the American Journal of Clinical Nutrition – also reveals that carbohydrate intake increased by 4% over the same period, while protein intake fell slightly. 

This is clear evidence that reducing a nation’s fat intake isn’t going to impact on weight problems. 

Fat does not make you fat – it’s too many refined carbohydrates and sugar that are the culprits. This is why eating a low-GL diet works so well, because it reduces intake of the harmful kinds of carbohydrate while increasing protein and healthy fats from sources like oily fish, nuts and seeds. This balances your blood sugar and helps you achieve, and maintain, a healthy weight. 

And speaking about too many refined carbs and sugar,
Check this out, only three foods make my cut: Steel Cut Oats, King Arthur organic flour, and quinoa.  The rest are JUNQUE!

The Healthiest Carbs In Your Supermarket

 

23 Februari 2011

Another Privacy Breach and HIPAA

Here is some current reporting in regard to HIPAA and your privacy.  For many years, since this legislation was enacted, we have warned that it does little to protect your medical records or your privacy.  Rather it has been almost an open door for all kinds of distribution, even to many not involved in health care. 

We suggest you ask your health insurance provider about the status of your medical records and get copies of everything for your own file.

February 22, 2011, 4:02 pm

Health insurer fined $4.3M for HIPAA violation

By Jason Millman
The federal health department is slapping a Maryland health insurer with a $4.3 million civil money penalty (CMP) for violating medical records rules. 
Cignet Health's failure to honor patients' requests for access to their medical records earned the Department of Health and Human Services’s first-ever CMP for a violation of the Health Insurance Portability and Accountability Act of 1996 (HIPAA) privacy rule.
The fine represents the Obama administration’s toughened enforcement of medical privacy laws. The 2009 stimulus package, which provided almost $30 billion to develop electronic health record systems, included boosted penalties for HIPAA violations.
According to HHS’s Office for Civil Rights (OCR), Cignet was fined $1.3 million for denying 41 patients access to their medical records between September 2008 and October 2009. The insurer was fined another $3 million for failing to cooperate with the OCR investigation.
“Ensuring that Americans’ health information privacy is protected is vital to our healthcare system and a priority of this Administration,” HHS Secretary Kathleen Sebelius said in a statement. “[HHS] is serious about enforcing individual rights guaranteed by the HIPAA Privacy Rule.” 

Daniel E. Austin, the owner of Cignet Health, a Christian-influenced health center, did not return calls to his office seeking comment. The Maryland Board of Physicians revoked his license in 2000 for his conviction for mail and loan fraud. Among the physicians listed on the center's Web site is one whose license was revoked in 2008 for engaging in sexual improprieties and sexual misconduct with patients.
Seeger said Cignet also provided health insurance. But last year, the Maryland Insurance Administration ordered Cignet to stop selling health insurance because it was not licensed to do so.
Several of the patients informed Cignet that they were requesting copies of their medical records so they could see doctors other than those working at Cignet, according to HHS documents.
To date HHS Secretary Sebelius has failed to enact several parts of healthcare legislation related to records due in Summer 2010.

Selections from Natural Health News
 
Oct 17, 2010
I started posting articles about electronic health and medical records in 2006 on Natural Health News. I am not in favour of this push in the arena of cost savings in the US health system. To date little has been shown to indicate any 
Jul 19, 2010
1 in 10 medical records on the new electronic database contains errors that could put patients at risk, doctors warn. They contain out of date information, errors on medication or drug allergies etc. ...
Jul 28, 2009
We also know that electronic medical record will not save money as well as the fact that HIPAA was the open sesame for everyone to get access to your data. You do have a choice, and most likely it is to find a health care professional
Nov 17, 2009
I started posting articles about electronic health and medical records in 2006 on Natural Health News. I am not in favour of this push in the arena of cost savings in the US health system. To date little has been shown to indicate any
May 21, 2008
Google's online filing cabinet for medical records opened to the public Monday, giving users instant electronic access to their health histories while reigniting privacy concerns. Called Google Health, the service lets users link ...

22 Februari 2011

Kombucha, For Real This Time

This SHOULD have been the Kombucha post that I posted last November. This SHOULD have been the only post I ever wrote about Kombucha. But the story took a weird, gross and kind of moldy turn after I so positively wrote about kombucha a few short months ago.

My first batch of kombucha tasted right. It was finished around New Years. But once bottled, my booch never carbonated. Curious.

Then my second batch molded. Fail

Then my third batch looked promising. A slight film developed on top. I waited patiently for this film to turn into a proper scoby. Oh the eager awaiting!!! After a week I noticed two small white spots. S#!t!! I fished out what was probably mold. I held my breath for another 2 weeks. No mold came back. I thought I was in the clear.

Then, after another few days, I saw something I thought looked right, a little pinkish ball forming on the side. It had a little arm that reached down into the tea. Here we go I thought. My first scoby!! The next day I noticed about a dozen small spots forming on the top. More mushrooms? Or mold? I wasn’t sure. I figured all would be revealed to me. After a couple days there was no fuzz growing off the spots so I thought they probably weren’t mold. But the strangest thing happened. The white spots had kind of attracted to one another like little funky magnets. Now instead of spread all over the top they were lined up like some kind of creepy firing line. My first reaction was to shudder a little. But I still wasn’t sure that it was mold. It wasn’t fuzzy. The booch still smelled right, like fruity fermenting sweetness. But I wasn’t so sure I wanted to drink this stuff.

A month or so ago a friend at work who reads the blog was laughing at me about my struggles with my moldy kombucha (in a nice way, you know). She suggested I call M because she said she had gotten her scoby from M. But I had never met M. I didn’t work with M in any capacity. It might be weird to call him out of the blue. So while I filed the suggestion in my brain, I wasn’t sure I would call him.

But I changed my mind when I saw a microscopic army forming in my kombucha. Was this army with me or against me? I picked up the phone and dialed M's desk. “You don’t know me”, I said “But I am C and I work in the New York Office. Someone told me you brew kombucha and I really need help.” He chuckled and told me that he had been brewing booch for over 10 years. I told him my woes, and he told me unequivocally-pitch my batch. It was likely mold. We talked about kombucha and fermentation for over 30 minutes. It was a slow day—I still got all my work done I PROMISE!! Then he kindly said that he would send me a scoby from his batch, which had never had mold. WooHoo! I had hit the jackpot! A few days later he emailed me to say that he had sent the scoby wrapped in plastic baggies and some paper, and then he put it in one of those standard issue yellow envelopes. And he popped that sucker through interoffice mail. Hahaha!


That’s right. My scoby took a trip through the UPS system and wound up on my desk the next afternoon. A healthy scoby should be thick and pinkish-brownish and have a faint vinegary smell. I held the envelope rather than slipping it in my work bag. My whole subway ride home I felt it through the paper. I imagined it warming from my touch. This thing really is alive I thought. I almost felt like I was adopting a pet, this scoby seemed to have its own aura. Perhaps I am over exaggerating. But I was so appreciative that M had taken the time to send it to me. I wanted my relationship with my kombucha to be positive from the get go.

I went home and tenderly unpacked it and washed it (took a picture). It was a half an inch thick, rubbery feeling and almost twice the diameter of the dehydrated scoby that I had bought online. My previous scoby was about 3 inches in diameter, whitish and about the thickness of 4-5 sheets of paper stacked together. I knew looking at my new friend that my days of worrying about my kombucha were over. I slipped it into a gallon of prepared sweet tea. Our relationship had begun. My moldy woes were over.
I get it now. You can’t buy a scoby online from a company. Kombucha is a living thing; it wants to build a relationship with you. And when you are starting out, you will have questions. If you don’t have someone to call you could drink moldy tea or get very sick. This kombucha is about relationships, between you and your scoby and you and your booch-mentor. I am pleased that M is my booch-mentor. He knows the lineage of my batch and he will be helpful should I have any questions. He knows how long this family of scobies takes to brew a batch and also had some helpful tips on prepping the tea.

Here are my OFFICIAL kombucha preparation instructions, as given to me by M.

1) Wash out your vessel with soap and hot water!! Make sure you start out with a clean glass gallon jar, be sure to wash off all the soap. Openings of 3 inches in diameter are good for easily getting your scoby in and out.
2) In a Dutch oven bring a gallon of water up to a boil. Boil at a rolling boil for 10 minutes to kill anything that might be in there.
3) Add one cup of organic sugar and boil until dissolved.
4) Turn off the heat and add 8 organic tea bags. Let stand for 30 minutes.
5) Let the batch cool for 10-12 hours. I poured my sweet tea into my completely clean glass jar to cool while I was at the office. But M said he lets his stand in his pot with the lid on the stove.
6) When it is totally cool and in the glass jar, add about 2-3 cups of prepared kombucha (I used a bottle of GT’s for my first batch), and float your scoby in there.
7) Let it brew, 10 days to 2 weeks. Taste the batch to see when you are happy with the sweet and sour balance.
8) Bottle in glass bottles with either a Grolsh style top, or I plan on using old cleaned out GTs bottle because the caps are sturdy and they close.
9) Come back here in 2 weeks and I will have more experience to talk about the second fermentation….

I am at peace. And finally I trust my booch. Thanks M!

Cholesterol: Another Wrong Conclusion

Poor Predictor: Researchers reported that the cholesterol levels in women were not associated with stroke, while there was only an association in men with levels higher than 9mmol/litre. The average in UK men is 5.5.

I don't know where people's heads have been since the cholesterol bonanza began, but my stand is that these pundits overlooked a known fact, which now seems to be getting press after some 30 years. 

It's triglycerides!  And this is what I learned in college, what will really hurt you and lead you along the slippery slope to heart attack and stroke.

I have often written that once milk began to be homogenized, things started going down hill. This practice by commercial dairies caused dairy fat to be "osmosed" directly into your blood, in fool fat form.  Clogged arteries are the end result, that s if course unless you follow a health promoting food plan or use proper supplementation.

The comes the low-fat/non-fat diet and all kinds of health problems reach new, high levels.  More drugs, less benefit, more cases, no recovery, lack of prevention too!
High levels of cholesterol do not predict the risk of stroke.
They did detect an increased risk in men, but only when cholesterol was at almost twice the average level.
The report in Annals of Neurology recommends using a different type of fat in the blood, non-fasting triglycerides, to measure the risk.
The Stroke Association said triglyceride tests needed to become routine to reduce the risk of stroke.
A total of 150,000 people have a stroke in the UK each year. Most are ischemic strokes, in which a clot in an artery disrupts the brain's blood supply.
The research followed 13,951 men and women, who took part in the Copenhagen City Heart Study.
During the 33-year study, 837 men and 837 women had strokes.
Poor predictor
They reported that the cholesterol levels in women were not associated with stroke, while there was only an association in men with levels higher than 9mmol/litre. The average in UK men is 5.5.
The researchers at Copenhagen University Hospital said this was "difficult to explain" as LDL, or bad, cholesterol is known to cause atherosclerosis which can block arteries.
They did notice a link, in both men and women, between the risk of stroke and non-fasting triglycerides.
They believe these fats are a marker for "remnant cholesterol" which is left behind when other forms of cholesterol are made.
Dr Peter Coleman, deputy director of research at The Stroke Association said: "Tests for triglyceride levels aren't routinely carried out in the UK unless there is significant concern."
"We know that high levels of fats, such as cholesterol, increase your risk of having a stroke. However, this research shows the importance of measuring the fat triglyceride, as well as cholesterol.
"This study highlights the importance of measuring triglycerides routinely in order to reduce a person's risk of stroke." http://www.bbc.co.uk/news/health-12505230


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